![]() ![]() ![]() Specific guidelines for treating PRD in such situations are lacking. Thus, these patients are predisposed to a higher risk of developing RD in the presence of PRD. Vitreous changes have been documented following refractive and cataract surgeries and other intraocular procedures. Since the past several decades, surgical correction of refractive errors, particularly myopia, is on the rise. The inherent demographic differences seen in the Indian population which shows increased prevalence of high myopia and myopia-related PRDs necessitates that we reconsider these guidelines as they apply to this population. The next best evidence is a consensus of expert opinion–based guidelines. A randomized controlled trial would be ideal but impractical considering the long follow-up time needed and the rarity of the occurrence. In a Cochrane systemic meta-analysis review published in 2014, Wilkinson attempted to assess the efficacy and need for preventive treatment for PRDs in the presence or absence of symptomatic PVD but failed to draw any conclusive evidence. Treatment of these lesions would be required to achieve prophylaxis against the development of RD. There are peripheral retinal lesions such as retinal breaks or lattice degeneration that can predispose to RD. Their subsequent management plays an important role in reducing the risk of RD, especially under special circumstances such as prior to refractive surgery or any intraocular procedure or in patients with abnormal vitreous such as in Sticker syndrome or Marfan syndrome. Thus, it is important to properly evaluate the retinal periphery for the presence of PRDs. This can lead to the development of retinal tears and cause RD. Anomalous, acute, symptomatic PVD with recent-onset flashes and floaters (within 3 months duration), in the presence or absence of PRD, can lead to various deleterious effects on the retina as a result of abnormal traction at the vitreoretinal interface. The development of PVD usually occurs either spontaneously in old age, as seen in most cases, or can be brought about by events such as cataract and refractive surgeries, intravitreal injections, retinal trauma, uveitis, pan-retinal photocoagulation, laser capsulotomy and syndromic diseases such as Marfan syndrome or Stickler syndrome. PVD is defined as the separation of the posterior cortical vitreous from the internal limiting membrane of the retina and is the most common cause of floaters. Posterior vitreous detachment (PVD) plays a crucial role in precipitating RD in eyes with PRDs. can result in rhegmatogenous retinal detachment (RD). While most of them are clinically insignificant, a few of these degenerations such as lattice degeneration, degenerative retinoschisis, peripheral retinal tears, cystic retinal tufts and, rarely, zonular traction tufts. Peripheral retinal degeneration (PRD) is a broad term which includes various lesions such as the lattice degeneration, snail-track degeneration, snowflake degeneration, atrophic or operculated retinal holes, peripheral retinal tears, senile retinoschisis, white and dark without pressure areas, paving stone degeneration and peripheral cystoid degeneration. ![]()
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